Clinical Chemistry
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


Clinical Chemistry 40: 1479-1484, 1994;
This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Milne, D. B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Milne, D. B.

Clinical Chemistry, Vol 40, 1479-1484, Copyright © 1994 by American Association for Clinical Chemistry

Assessment of copper nutritional status

DB Milne
US Department of Agriculture, Grand Forks Human Nutrition Research Center, ND 58202-9034.

Despite increased interest in the role of copper deficiency in clinical problems and an increased understanding of the physiological roles of copper, the diagnosis of a marginal deficiency has not been perfected. The use of non-standardized procedures and the effects of factors other than copper nutriture have impeded identification of the "ideal" indicator of copper nutritional status in adult humans. The specific activity of copper enzymes, or of copper-containing enzymes in blood cells, such as erythrocyte superoxide dismutase and platelet or leukocyte cytochrome c oxidase, may be a better indicator of metabolically active copper stores than the serum concentration of copper or ceruloplasmin, because the enzyme activities are sensitive to changes in copper stores and are not as sensitive to factors not related to copper nutriture. A single index, such as serum copper concentration, is inadequate for assessing the total body copper nutriture of an individual and must be supported by corroborating evidence.


The following articles in journals at HighWire Press have cited this article:


Home page
Postgrad. Med. J.Home page
P J Twomey
Effect of a different caeruloplasmin assay method on the relationship between serum copper and caeruloplasmin
Postgrad. Med. J., October 1, 2008; 84(996): 549 - 551.
[Abstract] [Full Text] [PDF]


Home page
Mayo Clin Proc.Home page
N. Kumar
Copper Deficiency Myelopathy (Human Swayback)
Mayo Clin. Proc., October 1, 2006; 81(10): 1371 - 1384.
[Abstract] [Full Text] [PDF]


Home page
Exp. Biol. Med.Home page
E. C. West and J. R. Prohaska
Cu,Zn-Superoxide Dismutase Is Lower and Copper Chaperone CCS Is Higher in Erythrocytes of Copper-Deficient Rats and Mice
Experimental Biology and Medicine, September 1, 2004; 229(8): 756 - 764.
[Abstract] [Full Text] [PDF]


Home page
J. Nutr.Home page
J. G. Penland and J. R. Prohaska
Abnormal Motor Function Persists Following Recovery from Perinatal Copper Deficiency in Rats
J. Nutr., August 1, 2004; 134(8): 1984 - 1988.
[Abstract] [Full Text] [PDF]


Home page
Am. J. Clin. Nutr.Home page
J. R Turnlund, R. A Jacob, C. L Keen, J. Strain, D. S Kelley, J. M Domek, W. R Keyes, J. L Ensunsa, J. Lykkesfeldt, and J. Coulter
Long-term high copper intake: effects on indexes of copper status, antioxidant status, and immune function in young men
Am. J. Clinical Nutrition, June 1, 2004; 79(6): 1037 - 1044.
[Abstract] [Full Text] [PDF]


Home page
J. Nutr.Home page
M. Hambidge
Biomarkers of Trace Mineral Intake and Status
J. Nutr., March 1, 2003; 133(3): 948S - 955.
[Abstract] [Full Text] [PDF]


Home page
J. Nutr.Home page
J. Bertinato, M. Iskandar, and M. R. L'Abbe
Copper Deficiency Induces the Upregulation of the Copper Chaperone for Cu/Zn Superoxide Dismutase in Weanling Male Rats
J. Nutr., January 1, 2003; 133(1): 28 - 31.
[Abstract] [Full Text] [PDF]


Home page
Clin. Chem.Home page
K. Minakata, O. Suzuki, and F. Horio
Quantification of Copper in Biological Materials by Use of Electron Spin Resonance,
Clin. Chem., October 1, 2001; 47(10): 1863 - 1865.
[Full Text] [PDF]


Home page
J. Am. Coll. Nutr.Home page
B. Bruce, G. A. Spiller, L. M, Klevay, and S. K. Gallagher
A Diet High in Whole and Unrefined Foods Favorably Alters Lipids, Antioxidant Defenses, and Colon Function
J. Am. Coll. Nutr., February 1, 2000; 19(1): 61 - 67.
[Abstract] [Full Text] [PDF]


Home page
J. Nutr.Home page
A. J. Fascetti, J. G. Morris, and Q. R. Rogers
Dietary Copper Influences Reproductive Efficiency of Queens
J. Nutr., December 1, 1998; 128(12): 2590S - 2590.
[Full Text]


Home page
Toxicol PatholHome page
A. Giovanetti, L. Rossi, M. Mancuso, C. C. Lombardi, M. R. Marasco, F. Manna, P. Altavista, and E. M. Massa
Analysis of Lung Damage Induced by Trichloroethylene Inhalation in Mice Fed Diets with Low, Normal, and High Copper Content
Toxicol Pathol, September 1, 1998; 26(5): 628 - 635.
[Abstract] [PDF]


Home page
Arterioscler. Thromb. Vasc. Bio.Home page
W. Y. Craig, S. E. Poulin, G. E. Palomaki, L. M. Neveux, R. F. Ritchie, and T. B. Ledue
Oxidation-Related Analytes and Lipid and Lipoprotein Concentrations in Healthy Subjects
Arterioscler Thromb Vasc Biol, June 1, 1995; 15(6): 733 - 739.
[Abstract] [Full Text]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1994 by the American Association for Clinical Chemistry.