Clinical Chemistry Link to Randox Laboratories Web Site
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

Clinical Chemistry 45: 1347-1352, 1999;
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Submit an electronic Letter to
the Editor about this paper
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via ISI Web of Science (1)
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Mundy, G. R.
Right arrow Articles by Guise, T. A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Mundy, G. R.
Right arrow Articles by Guise, T. A.
Related Collections
Right arrow Arnold O. Beckman Conference
(Clinical Chemistry. 1999;45:1347-1352.)
© 1999 American Association for Clinical Chemistry, Inc.

Articles

Hormonal Control of Calcium Homeostasis

Gregory R. Mundya and Theresa A. Guise

University of Texas Health Science Center, Medicine/Endocrinology, 7703 Floyd Curl Dr., San Antonio, TX 78284-7877.
a Author for correspondence. Fax 210-567-6693; e-mail mundy{at}uthscsa.edu

Calcium homeostasis in the extracellular fluid is tightly controlled and defended physiologically. Hypercalcemia always represents considerable underlying pathology and occurs when the hormonal control of calcium homeostasis is overwhelmed. The major hormones that are responsible for normal calcium homeostasis are parathyroid hormone and 1,25-dihydroxyvitamin D; these hormones control extracellular fluid calcium on a chronic basis. Over- or underproduction of these hormones or the tumor peptide, parathyroid hormone-related peptide, are the major causes of aberrant extracellular fluid calcium concentrations. These hormonal defense mechanisms are reviewed here.© 1999 American Association for Clinical Chemistry




The following articles in journals at HighWire Press have cited this article:


Home page
 Am. J. Physiol. Regul. Integr. Comp. Physiol.Home page
P. M. Guerreiro, J. L. Renfro, D. M. Power, and A. V. M. Canario
The parathyroid hormone family of peptides: structure, tissue distribution, regulation, and potential functional roles in calcium and phosphate balance in fish
Am J Physiol Regulatory Integrative Comp Physiol, February 1, 2007; 292(2): R679 - R696.
[Abstract] [Full Text] [PDF]

Home page
 IBMS BoneKEyHome page
D. B. Henriksen
The Gut Feeling of Bone Remodeling
IBMS BoneKEy, November 1, 2005; 2(11): 16 - 23.
[Full Text] [PDF]

Home page
 Endocr Relat CancerHome page
G A Clines and T A Guise
Hypercalcaemia of malignancy and basic research on mechanisms responsible for osteolytic and osteoblastic metastasis to bone
Endocr. Relat. Cancer, September 1, 2005; 12(3): 549 - 583.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Clin. Nutr.Home page
M. Cifuentes, C. S Riedt, R. E Brolin, M P. Field, R. M Sherrell, and S. A Shapses
Weight loss and calcium intake influence calcium absorption in overweight postmenopausal women
Am. J. Clinical Nutrition, July 1, 2004; 80(1): 123 - 130.
[Abstract] [Full Text] [PDF]

Home page
 CLIN PEDIATRHome page
V. Umpaichitra, W. Bastian, and S. Castells
Hypocalcemia in Children: Pathogenesis and Management
Clinical Pediatrics, June 1, 2001; 40(6): 305 - 312.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1999 by the American Association for Clinical Chemistry.