Clinical Chemistry
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Clinical Chemistry 55: 1909, 2009; 10.1373/clinchem.2009.133777
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(Clinical Chemistry. 2009;55:1909.)
© 2009 American Association for Clinical Chemistry, Inc.

Commentaries

Commentary

Robert Richardson

Toronto General Hospital, Toronto, ON, Canada.

Address correspondence to the author at: Toronto General Hospital, 200 Elizabeth St., Toronto, ON, Canada. Fax 416-340-4999; e-mail robert.richardson@uhn.on.ca.

The first 20% of the full text of this article appears below.

Patients with chronic hyponatremia have vasopressin acting on the kidney, causing water retention and dilution of the sodium in the extracellular fluid. In surveys of hospitalized patients with hyponatremia, the stimulus for vasopressin secretion is low effective circulating volume via the baroreceptor in about two-thirds of patients. . . . [Full Text of this Article]






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Copyright © 2009 by the American Association for Clinical Chemistry.